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Chen Tian Hua
Cardiology
About me
Cardiologist, Associate Chief Physician, has been engaged in cardiovascular clinical work for 20 years, accumulating rich clinical experience.
Proficient in diseases
Diagnosis and treatment of various difficult and critically ill cardiovascular diseases.
Voices
How is pericardial effusion treated?
The treatment of pericardial effusion mainly includes the following measures: First, treatment should be based on the different causes of pericardial effusion. For example, pericardial effusion caused by infection should be treated with anti-infection measures, and pericardial effusion caused by heart failure should be treated with anti-heart failure measures; Second, for a large amount of pericardial effusion or pericardial effusion causing cardiac tamponade, active pericardiocentesis and drainage should be provided to relieve the pressure on the heart caused by the pericardial effusion; Third, when severe pericardial effusion causes hypotension or even shock, volume expansion and pressor agents should also be administered; Fourth, for some patients with pericardial effusion, if the absorption of the effusion is not effective after general treatment, corticosteroids may be appropriately administered.
Ventricular premature beat
Ventricular premature beats refer to heartbeats that occur prematurely below the bifurcation of the bundle of His, causing early depolarization of the myocardium; they are a common arrhythmia in clinical settings. Ventricular premature beats can occur in healthy individuals and may be triggered by factors such as fatigue and staying up late, excessive smoking and drinking, excessive mental stress, and consuming strong tea and coffee. They can also be seen in organic heart diseases such as coronary artery disease, heart failure, hypertensive heart disease, myocarditis, and cardiomyopathy. For ventricular premature beats, it is crucial to actively remove the related triggers. If accompanied by organic heart disease, it is necessary to actively control the underlying disease.
What is mild mitral and tricuspid regurgitation of the heart?
Mild regurgitation of the mitral and tricuspid valves means that during the contraction phase of the heart, the mitral and tricuspid valves cannot close completely, resulting in a slight imperfection in closure. This causes a small amount of blood to flow back from the ventricle into the atrium. Since the closure imperfection is minor, the volume of the regurgitating blood is not significant. If not accompanied by any organic disease, this type of regurgitation typically does not lead to adverse outcomes and does not require treatment. However, if the patient has an organic disease, especially organic heart disease, it is crucial to actively control the related diseases to prevent aggravation, which could worsen the condition of mitral and tricuspid valves regurgitation, leading to more severe regurgitation.
Can mitral and tricuspid regurgitation heal by themselves?
Mitral and tricuspid valve regurgitation generally cannot heal on their own. Of course, different cases of mitral and tricuspid regurgitation require different clinical treatments. Generally speaking, if it is only mild mitral and tricuspid regurgitation, no special treatment is needed. If it is caused by organic lesions in the mitral and tricuspid valves themselves, causing severe regurgitation, then surgical treatment is necessary to correct the organic abnormalities of the valves. If the regurgitation of the tricuspid and mitral valves is caused by diseases that lead to an enlarged heart and dilation of the valve annulus, it is necessary to actively treat these diseases to effectively control the condition and prevent it from worsening.
What should I do if I have mild mitral valve regurgitation?
Mild mitral valve regurgitation is observed and further investigation is needed to understand the causes of this condition. If mild mitral valve regurgitation occurs in a healthy individual, it is primarily due to physiological degeneration of the mitral valve, generally does not lead to adverse consequences, and does not require special treatment. If it is caused by diseases that lead to an enlarged heart, resulting in relative mild mitral valve regurgitation, this situation necessitates active treatment of the underlying diseases to effectively control the condition, prevent further enlargement of the heart, exacerbate the degree of mitral valve regurgitation, and thus adversely affect health.
What is mild regurgitation of the mitral and tricuspid valves?
If a cardiac echocardiogram simply finds mild regurgitation of the mitral and tricuspid valves, and the heart isn't noticeably enlarged, and the valves have no organic lesions, this mild regurgitation of the mitral and tricuspid valves isn't considered a pathological state. It should be considered as physiological degeneration of the valves and doesn't require treatment, generally causing no harm to overall health. However, some occurrences of mild regurgitation of the mitral and tricuspid valves are due to enlargement of the left and right ventricles. In these cases, it's necessary to further identify the causes leading to the enlargement of the ventricles and actively address them to control the condition effectively. This avoids further enlargement of the ventricles, which could worsen the valve regurgitation and negatively impact health.
How is acute pericarditis classified?
Acute pericarditis is classified according to the course of the disease, including acute fibrinous pericarditis and acute exudative pericarditis. If classified according to the cause, it can be divided into infectious pericarditis and non-infectious pericarditis. Infectious pericarditis can be caused by viruses, bacteria, tuberculosis, fungi, etc., while non-infectious pericarditis can be seen in tumors, uremia, acute myocardial infarction, aortic dissection, connective tissue disease, trauma, and cardiac surgery, etc. Acute pericarditis is an acute inflammatory disease of the pericardium's parietal layer, and its occurrence requires timely diagnosis. It is also important to further clarify the specific cause of acute pericarditis and actively treat it.
Ventricular septal defect is what?
Ventricular septal defect is a common congenital heart disease, mainly due to the abnormal defect in the ventricular septum during the fetal development, resulting in an abnormal channel between the left and right ventricles. Ventricular septal defect can cause blood to shunt from the left ventricle to the right ventricle, gradually increasing the blood flow in the pulmonary circulation, leading to an increase in the volume load of the left and right ventricles, while causing a decrease in the systemic circulation volume. In the later stages, due to the significant increase in right ventricular pressure, it leads to right-to-left shunting, thereby causing Eisenmenger's syndrome. For ventricular septal defects with a large degree of defect, timely interventional treatment or surgical ventricular septal defect repair is required.
What to do with mild mitral and tricuspid regurgitation?
The appearance of mild regurgitation in the mitral and tricuspid valves requires further clarification of the specific causes leading to the regurgitation, and treatment should be based on these causes. In healthy individuals, mild regurgitation of the mitral and tricuspid valves may be considered physiological degeneration, which is not considered a disease and does not require special treatment. However, mild regurgitation of these valves can also occur due to related organic diseases causing ventricular enlargement and annular dilation, leading to relative mild regurgitation of the mitral and tricuspid valves. In such cases, effective treatment of the underlying disease is necessary to keep it stable in the long term, preventing further enlargement of the ventricle that could worsen valve regurgitation and adversely affect the patient.
What are the dangers of ventricular premature beats?
The hazards of ventricular premature beats can be mainly divided into the following aspects: First, if a healthy person experiences ventricular premature beats, it can cause symptoms such as chest tightness, palpitations, fatigue, and dizziness. If these symptoms are quite noticeable, they often affect the patient's work and life, and at night, they can also impact sleep, leading to sleep deprivation; Second, if the patient has severe structural heart disease, frequent ventricular premature beats can induce angina attacks in patients with coronary artery disease, and can lead to worsening of heart failure in patients suffering from heart failure; Third, some malignant ventricular premature beats can also induce severe rapid malignant arrhythmias, such as sustained ventricular tachycardia and ventricular fibrillation, leading to sudden cardiac arrest and sudden cardiac death.