What are the harms of hyperuricemia?

Written by Li Hui Zhi

Endocrinology

Updated on August 31, 2024

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The dangers of hyperuricemia:

Firstly, some individuals might experience gouty arthritis, characterized by localized joint swelling, redness, heat, and pain, affecting mobility.

Secondly, prolonged hyperuricemia can damage the kidneys, leading to chronic renal failure.

Thirdly, if hyperuricemia persists long-term, it may result in the formation of tophi. Tophi deposited in joints can cause joint deformities and limited mobility. Additionally, hyperuricemia is an independent risk factor for cardiovascular and cerebrovascular diseases, and thus should be taken seriously.

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Written by Luo Juan

Endocrinology

57sec

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What fruits to eat for hyperuricemia?

For patients with hyperuricemia, we generally need to limit the intake of high-purine foods, such as crab and animal offal. Moderate consumption of medium-purine foods, such as meats and fish, is acceptable. Low-purine foods like vegetables, fruits, milk, and eggs can be safely consumed. For example, fruits like grapes, pears, grapefruits, kiwis, and dragon fruits are all acceptable to eat. However, many patients with hyperuricemia also have other metabolic abnormalities, such as high blood sugar. In such cases, it is important to monitor the quantity of fruit intake because excessive consumption can lead to fluctuations in blood sugar levels. Therefore, fruits can be safely consumed by those with hyperuricemia, but attention must be paid to any additional metabolic abnormalities that may be present.

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Written by Li Jing

Rheumatology

1min 17sec

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Are hyperuricemia and high uric acid the same thing?

Firstly, hyperuricemia and high uric acid levels refer to the same concept, defined as blood uric acid levels greater than 420. This condition is described as asymptomatic hyperuricemia, which does not require special treatment at this stage, but it does necessitate changes in lifestyle habits such as abstaining from alcohol, avoiding eating animal organs, seafood, and high-protein foods, not drinking carbonated beverages, and losing weight if obese, to help control body weight. After changing these lifestyle habits, a recheck of the uric acid levels should be done in two weeks to see if there has been a decrease. If the levels have not decreased at that time, sodium bicarbonate tablets can be used to alkalinize the urine. Additionally, increasing water intake to promote the excretion of uric acid and monitoring the dynamic changes in uric acid levels are needed. Once joint pain occurs, which indicates an acute gout attack, proper uric acid-lowering medication is required. (Please undergo medication under the guidance of a professional physician, and do not self-medicate blindly.)

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Written by Luo Juan

Endocrinology

46sec

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Can people with hyperuricemia eat wood ear mushrooms?

People with hyperuricemia can eat wood ear mushrooms. As wood ear mushrooms are considered a low-purine food, containing approximately 8.8mg of purines per 100 grams, they can be consumed during hyperuricemia. For hyperuricemia, it is necessary to restrict some high-purine foods, such as animal offal, clams, crabs, oysters, and sardines. Some meats, seafood, peas, and spinach, which also contain a certain amount of purines, can be consumed in moderation. Wood ear mushrooms are a low-purine food and are generally not restricted. Thus, people with hyperuricemia can eat wood ear mushrooms.

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Written by Li Hui Zhi

Endocrinology

29sec

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Can people with hyperuricemia drink alcohol?

Patients with hyperuricemia are advised to avoid drinking alcohol. Especially liquors such as baijiu and beer, which have high purine content, are contraindicated for patients with hyperuricemia. Furthermore, if hyperuricemia is diagnosed with high levels of uric acid, one should abstain from alcohol, and also pay attention to diet by avoiding foods high in purines to prevent complications such as gouty arthritis and gouty kidney.

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Written by Lin Xiang Dong

Endocrinology

39sec

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Causes of hyperuricemia

Hyperuricemia is divided into primary hyperuricemia and secondary hyperuricemia. Primary hyperuricemia is mainly due to a disorder in the metabolism of purines in the body, leading to excessive production of uric acid, which in turn causes hyperuricemia. Prolonged duration can greatly increase the risk of gout. Secondary hyperuricemia refers to hyperuricemia caused by excessive production or excretion obstacles due to some common diseases, the most common being chronic renal failure or some tumors, etc.