Can people with hyperuricemia eat wood ear mushrooms?

Written by Luo Juan
Endocrinology
Updated on November 13, 2024
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People with hyperuricemia can eat wood ear mushrooms. As wood ear mushrooms are considered a low-purine food, containing approximately 8.8mg of purines per 100 grams, they can be consumed during hyperuricemia. For hyperuricemia, it is necessary to restrict some high-purine foods, such as animal offal, clams, crabs, oysters, and sardines. Some meats, seafood, peas, and spinach, which also contain a certain amount of purines, can be consumed in moderation. Wood ear mushrooms are a low-purine food and are generally not restricted. Thus, people with hyperuricemia can eat wood ear mushrooms.

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Written by Luo Juan
Endocrinology
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High uric acid blood disease falls under which department?

Hyperuricemia is an endocrinological disease. First, let's understand what hyperuricemia is. Hyperuricemia refers to a condition where the concentration of uric acid in the blood exceeds the normal upper limit. Generally, the blood uric acid concentration in males and postmenopausal females is greater than 420 µmol/L, and greater than 348 µmol/L in premenopausal females is considered hyperuricemia. It is a nucleic acid metabolic disease and is often accompanied by obesity, type 2 diabetes, hyperlipidemia, hypertension, arteriosclerosis, coronary heart disease, etc., and is a type of metabolic disorder. Therefore, hyperuricemia is an endocrinological disease.

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Written by Chen Xie
Endocrinology
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Can hyperuricemia be cured?

Hyperuricemia is a metabolic disease caused by disorders in purine metabolism and is clinically divided into primary and secondary categories. Primary hyperuricemia is caused by congenital abnormalities in purine metabolism and is often associated with obesity, dyslipidemia, hypertension, atherosclerosis, and coronary heart disease. Secondary hyperuricemia, on the other hand, is caused by certain systemic diseases or medications. Therefore, whether hyperuricemia can be cured depends on the underlying condition, and the efficacy of treatment cannot be generalized. For hyperuricemia caused by medications or systemic diseases, it can be cured by treating the disease or discontinuing the medication. However, for hyperuricemia caused by congenital abnormalities in purine metabolism, it can be clinically managed by controlling diet and medication, but it is not curable.

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Written by Lin Xiang Dong
Endocrinology
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Hyperuricemia manifestations

Most cases of hyperuricemia usually have no obvious clinical symptoms, and it can take years to decades from the increase in blood uric acid to symptom onset, with some individuals never exhibiting obvious clinical symptoms throughout their lifetime. However, as age increases, the incidence of gout becomes higher. When accompanied by acute gouty arthritis, patients may experience severe joint pain. If blood uric acid levels remain high over a long period, this can lead to the formation of gouty tophi and uric acid stones, among other issues.

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Written by Li Hui Zhi
Endocrinology
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Can people with hyperuricemia drink alcohol?

Patients with hyperuricemia are advised to avoid drinking alcohol. Especially liquors such as baijiu and beer, which have high purine content, are contraindicated for patients with hyperuricemia. Furthermore, if hyperuricemia is diagnosed with high levels of uric acid, one should abstain from alcohol, and also pay attention to diet by avoiding foods high in purines to prevent complications such as gouty arthritis and gouty kidney.

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Written by Chen Xie
Endocrinology
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How is hyperuricemia treated?

The treatment of hyperuricemia mainly includes: First, improving lifestyle, including healthy eating with a low-purine diet, quitting smoking, drinking more water, exercising regularly, and controlling weight. Second, alkalinizing the urine using sodium bicarbonate to maintain urine pH between 6.2 and 6.9, which facilitates the excretion of uric acid. Third, avoiding medications that increase blood uric acid levels, such as diuretics, corticosteroids, and insulin, among others. Fourth, using medications that lower uric acid, including drugs that increase uric acid excretion, mainly benzbromarone and probenecid, and drugs that inhibit uric acid synthesis, such as allopurinol and febuxostat. The choice of medication has specific indications, contraindications, and side effects, and it is advised to use these medications under the guidance of a specialist and not to self-medicate.