What are the symptoms of cerebral hemorrhage?

Written by Tang Li Li
Neurology
Updated on September 20, 2024
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Patients with cerebral hemorrhage typically experience sudden headaches during physical activity or emotional excitation as their initial symptom, which may be accompanied by nausea and vomiting. In severe cases, vomiting can be projectile, consisting of stomach contents. If the patient has stress ulcers leading to bleeding and erosion of the gastric mucosa, they may vomit a coffee-ground-like liquid, which is a mixture of gastric juices and blood. Additionally, patients may also exhibit disturbances in consciousness, such as drowsiness, stupor, or even coma. If the patient is conscious, one may observe hemiplegia or paralysis, sometimes accompanied by speech impairments or psychiatric symptoms. Some patients may also experience epileptic seizures.

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Causes of cerebral hemorrhage

The most common cause of cerebral hemorrhage is hypertensive cerebral hemorrhage. Patients with hypertension who do not adequately control their blood pressure, which frequently fluctuates, can lead to hardening of the cerebral vessels. The elasticity of the cerebral vessel walls diminishes, and when the blood pressure rises sharply, it can cause the cerebral vessels to rupture, leading to cerebral hemorrhage. This is the most common cause of cerebral hemorrhage, accounting for over 80%. Another common cause of cerebral hemorrhage is the rupture of cerebral aneurysms, which often leads to subarachnoid hemorrhage. Patients with this condition experience severe headaches, consciousness disturbances, and restlessness. Subarachnoid hemorrhage is a relatively severe medical condition with a high mortality rate. Additionally, some cerebral hemorrhages are caused by trauma, leading to cerebral contusion, skull fractures, subdural hematoma, and epidural hematoma. These causes account for a relatively small proportion of clinical cases.

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Complications of Cerebral Hemorrhage

The main complications of cerebral hemorrhage are infections or fever, especially aspiration pneumonia, which is quite common. The second complication is hyperglycemia. The third is stress ulcers and gastrointestinal bleeding. The fourth complication involves cardiac issues, as patients with cerebral hemorrhage are prone to neurogenic cardiac damage. Another complication is epilepsy; patients with lobar hemorrhage are likely to experience recurrent seizures. Additionally, there is also central fever.

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Written by Chen Ya
Geriatrics
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How to recover from cerebral hemorrhage

The recovery from cerebral hemorrhage is divided into the acute phase and the non-acute phase. In the acute phase, if there is a large amount of bleeding and increased intracranial pressure, surgical intervention can be considered. If the bleeding is less but intracranial pressure is still high, general treatment includes managing blood pressure well, maintaining it at 140 systolic pressure, under which conditions things generally tend to be better. Additionally, controlling intracranial pressure and treatment to stop the bleeding are needed. Later stages involve anticoagulant reversal treatments and prevention of complications because infections, high blood sugar, and stress ulcers can further worsen the condition. The later stages primarily focus on rehabilitation training treatments, helping with limb and speech rehabilitation training.

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Written by Tang Li Li
Neurology
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Does cerebral hemorrhage have a genetic basis?

Cerebral hemorrhage is not hereditary and is not a genetic disease; it is a type of acute cerebrovascular disease. Its occurrence is related to risk factors of cerebrovascular disease, and some risk factors may be related to genetics. Therefore, having a family history of cerebral hemorrhage is considered one of the factors that can increase the incidence rate of cerebral hemorrhage in patients. For example, if a patient's parents have hypertension, diabetes, or hyperlipidemia, these risk factors can significantly increase the likelihood that the patient will also develop these conditions as they age. Long-term hypertension, diabetes, and hyperlipidemia can lead to arterial wall hardening, sometimes resulting in hyaline degeneration. When blood pressure fluctuates, it can easily form microaneurysms that rupture and bleed, thus causing cerebral hemorrhage. Therefore, although cerebral hemorrhage itself is not hereditary, hereditary-related conditions such as hypertension and diabetes might lead to its occurrence.

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Written by Zhang Hui
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What can be eaten with cerebral hemorrhage?

Intracerebral hemorrhage is an extremely dangerous neurological disease. Its onset is very sudden, with patients experiencing headaches, nausea, vomiting, as well as symptoms like limb paralysis, numbness, and speech impairments. Additionally, the gastrointestinal function of patients is significantly affected during an intracerebral hemorrhage, making them very susceptible to acute ulcers and acute gastric mucosal lesions. Hence, there are some precautions to consider regarding diet. In the acute phase of intracerebral hemorrhage, it is recommended to eat softer, easily digestible foods, such as rice porridge, millet porridge, sesame paste, etc., which are easier to digest and can help reduce the burden on the gastrointestinal tract. After the acute phase, the patient should consume more fresh vegetables and fruits, which can help maintain regular bowel movements, beneficial for the recovery from intracerebral hemorrhage. These also provide a rich source of vitamins that can protect vascular endothelial cells and reduce the recurrence of intracerebral hemorrhage. Additionally, eating high-quality proteins such as drinking milk, eating eggs, and lean meats can help enhance the patient's resistance and reduce complications.