What should I do if cirrhosis causes general weakness?

Written by Wu Hai Wu

Gastroenterology

Updated on November 26, 2024

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Liver cirrhosis can lead to general weakness due to electrolyte disorders or severe liver function impairment, causing the symptoms mentioned above. Once general weakness occurs, it is necessary to go to the hospital for comprehensive auxiliary examinations, such as liver function tests, routine blood tests, and electrolyte tests. Additionally, imaging studies of the abdomen should be performed to determine whether conditions like ascites, anemia, or hyponatremia exist. Different treatments are administered based on different disease diagnoses. For instance, if the weakness is due to severe ascites causing dilutional hyponatremia, sodium supplementation might be necessary, along with appropriate ascites drainage.

Other Voices

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Written by Si Li Li

Gastroenterology

1min 8sec

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Can severe cirrhosis be cured?

Severe cirrhosis is understood to refer to the decompensated stage of cirrhosis, which is the later stage of the condition, primarily characterized by the hardening of the liver. Additionally, it presents a series of bodily complications such as ascites, as well as esophageal and gastric varices caused by portal hypertension. Sometimes, because of these varices, accidental rupture can occur during eating, leading to severe bleeding, which is a major upper gastrointestinal hemorrhage — a very dangerous complication. If this bleeding is not controlled, it can become life-threatening. Moreover, the skin may display spider nevi, and palmar erythema can appear on the hands; these are common manifestations and complications during the decompensated phase of cirrhosis. Thus, cirrhosis is not curable. Treatment focuses on symptomatic relief to minimize discomfort caused by the symptoms, but the disease itself cannot be cured.

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Written by Li Qiang

Intensive Care Unit

1min 9sec

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Is late-stage liver cirrhosis with liver failure easy to control?

In the late stages of cirrhosis, when the condition has progressed to liver failure, it becomes very difficult to manage. At this stage, due to liver failure, the first issue to arise is abnormal coagulation function, such as easy bleeding in patients. This will lead to severe hypoalbuminemia, causing swelling throughout the body. Additionally, bilirubin levels are very high, leading to various metabolic diseases, such as metabolic encephalopathy, which affects other organs as well. In the late stages of cirrhosis, there is also a large amount of ascites. At this time, portal hypertension is common, leading to complications such as gastric and esophageal varices rupture, bleeding, and hepatic encephalopathy, making the condition very difficult to control. If the cirrhosis is a benign lesion, at this time, adjusting liver function through methods such as artificial livers or plasma exchange can improve the condition to a manageable extent. It is critical to act quickly to perform a liver transplant, as there are no other effective treatments besides transplant, and the condition is very difficult to control.

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Written by Yang Chun Guang

Gastroenterology

40sec

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Is alcoholic cirrhosis ascites serious?

Once ascites caused by alcoholic liver cirrhosis is discovered, it indicates that the liver has reached the decompensated stage of cirrhosis, which is quite severe. Decompensated cirrhosis itself has a relatively low five-year and ten-year survival rate. Moreover, once the liver enters the decompensated stage, it is also prone to corresponding complications, such as gastrointestinal bleeding, hepatic encephalopathy, infections, etc. Therefore, when alcoholic liver cirrhosis presents with ascites, it is crucial to seek treatment at a formal hospital. The condition is quite serious and requires active treatment.

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Written by Huang Ya Juan

Gastroenterology

2min 16sec

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Early symptoms of cirrhosis

Liver cirrhosis is generally divided into compensated and decompensated stages: The compensated stage is actually an early stage of liver cirrhosis. During this stage, most patients do not show symptoms, or they may experience some mild symptoms such as abdominal discomfort, fatigue, loss of appetite, abdominal bloating, indigestion, diarrhea, etc. Decompensated stage: Early stages often present with fatigue and weakness, and later stages can include weight loss, loss of appetite, aversion to oily foods, abdominal bloating, diarrhea, fever, etc.; The liver is the only site of albumin synthesis, and during cirrhosis, reduced serum albumin can cause edema and ascites; the liver synthesizes most clotting factors, and cirrhosis can lead to coagulation disorders, clinically manifesting as gum bleeding, skin bruising, and nosebleeds; During cirrhosis, there can also be metabolic disorders of bilirubin and bile acids, leading to jaundice; Cirrhosis can also cause hormonal metabolic abnormalities, especially in patients with alcoholic cirrhosis, who may exhibit many characteristics of male dysfunction, including impotence, erectile dysfunction, testicular atrophy, loss of body hair and muscle mass, feminization, etc. Female patients may experience cessation or reduction of menstruation, excessive menstruation, infertility, etc.; Both male and female patients can develop osteoporosis, leading to spontaneous fractures.

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Written by Shen Jiang Chao

Radiology

48sec

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Can cirrhosis be detected by a CT scan?

Cirrhosis can be detected by a CT scan. Early-stage cirrhosis has no specific diagnostic value on CT, but middle to late-stage cirrhosis typically manifests as wavy changes in the liver capsule, enlarged liver lobes, and disproportionate size of liver lobes, with most changes involving an enlargement of the left lobe and caudate lobe and a relative shrinkage of the right lobe. Additionally, secondary manifestations of cirrhosis, such as portal hypertension, can cause enlargement of the spleen and varices in the lower esophagus and stomach fundus. When cirrhosis is detected, it is advisable to perform an enhanced CT scan to determine the potential for malignant transformation in liver regenerative nodules.