Which department should I go to for rickets?

Written by Li Jiao Yan
Neonatology
Updated on September 20, 2024
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Rickets, known as nutritional vitamin D deficiency rickets, is a chronic nutritional disease characterized by skeletal lesions, caused by a deficiency of vitamin D in the body leading to disturbed calcium and phosphate metabolism. In the early stages of rickets, it is primarily due to insufficient levels of vitamin D, which then leads to abnormal calcium and phosphate metabolism; at this stage, it is necessary to consult the endocrinology department to correct it timely. If rickets has already caused developmental deformities of the bones, orthopedic consultation is required for corrective treatment. For instance, if children exhibit pigeon chest, or noticeable bow legs or knock knees, they need to see an orthopedic specialist.

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Written by Quan Xiang Mei
Pediatrics
1min 6sec home-news-image

What should I do about the skull protrusion caused by rickets?

When children suffer from rickets with noticeable cranial protrusions and abnormal skeletal changes, it is crucial to timely supplement based on the specific needs of the child for trace elements like calcium, as well as vitamin D. Supplementation of vitamin D and trace elements such as calcium, combined with daily activities like sun exposure, are advisable. Additionally, integrating diet changes by consuming foods rich in calcium supports calcium and vitamin D absorption. Consequently, the cranial protrusions can gradually alleviate or disappear. As supplementation continues and the child ages, the shape of the skull may also change accordingly. In daily life, it is important to prevent skeletal abnormalities in children due to calcium and vitamin D deficiencies by making timely adjustments. (Note: The use of medications should be under the guidance of a professional doctor.)

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Written by Li Jiao Yan
Neonatology
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What is the treatment method for rickets?

Rickets is primarily caused by a deficiency in vitamin D, leading to disturbances in calcium and phosphorus metabolism in the body, which results in abnormal skeletal development—a type of systemic nutritional disease. The most common form is nutritional rickets due to a deficiency in nutrients and vitamin D. Treatment focuses on controlling the active phase to prevent relapse of deformities. Therefore, early detection and comprehensive treatment are crucial. The primary treatment method involves supplementing with vitamin D. The choice of vitamin D, the dosage, and the length of treatment generally need to be tailored to the specific circumstances of the child. Additionally, increased exposure to sunlight and appropriate outdoor activities should be encouraged to enhance the absorption of vitamin D. Children should also be supplemented with calcium and a variety of vitamins. Follow-ups during the recovery phase are necessary, and training in sitting, standing, and walking should not be started too early to avoid deformities in the lower limbs. Furthermore, no medication is needed during the residual stage of the disease. For mild to moderate cases, physical exercise should be intensified, and skeletal deformities can be corrected using active or passive exercises. Severe skeletal deformities may require surgical correction.

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Written by Li Jiao Yan
Neonatology
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Can rickets be inherited by the next generation?

Rickets, called vitamin D-deficiency rickets, is caused by a lack of vitamin D which leads to abnormal calcium and phosphorus metabolism in the body. This prevents calcium salts from properly depositing in the growing parts of the skeleton, resulting in incomplete mineralization of bone tissue during growth. It produces a systemic, chronic nutritional disease characterized by skeletal lesions and closely related to lifestyle. The primary causes of rickets are due to a combination of factors leading to vitamin D deficiency. Feeding methods, cod liver oil supplementation, living environment, time spent outdoors, recurrent respiratory infections, maternal calcium deficiency during pregnancy, and the season of birth are all significant contributing factors to the incidence of rickets. Generally, most cases of rickets are not inherited to the next generation. However, if there is a clear hereditary disease causing insufficient levels of vitamin D, it could potentially be inherited by the next generation.

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Written by Zeng Hai Jiang
Pediatrics
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Rickets and osteoporosis are caused by a lack of what?

Rickets and osteoporosis are both caused by a lack of vitamin D and calcium. Rickets is commonly seen in infants and can present with abnormalities in skeletal development such as "ping-pong ball head", signs of wrist and ankle bracelets, square skull, funnel chest, pigeon chest, X-shaped legs, and O-shaped legs. Osteoporosis is a metabolic bone disease caused by reduced bone formation and increased bone resorption. It can be categorized according to its etiology into senile osteoporosis, postmenopausal osteoporosis, idiopathic osteoporosis, and secondary osteoporosis. Clinically, it can manifest as fatigue, bone pain, and fractures.

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Written by Tong Peng
Pediatrics
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Will the frontal bossing from rickets get better?

Rickets, if presenting with frontal bossing, can recover depending on the specific condition and age of the child. Rickets is often divided into four stages, with sequelae usually occurring after the age of three. Therefore, if symptoms of rickets are present before the age of three, they can be alleviated through supplementation with vitamin D, calcium, and other trace elements. Symptoms of rickets include craniotabes, Harrison's groove, beading of the ribs, pigeon chest, everted ribs, bowlegs, and knock-knees. Frontal bossing is often a manifestation of craniotabes, which usually occurs during infancy. Supplementing with vitamin D during this period can greatly alleviate the condition. At the same time, it is important to also expose the child to sunlight and engage in outdoor activities to promote the bones' absorption of vitamin D and calcium, preventing the recurrence of symptoms.