Myasthenia gravis is an autoimmune disease that primarily affects the neuromuscular junction. It is an acquired autoimmune disorder in which acetylcholine receptors at the postsynaptic membrane are present. These receptors bind with acetylcholine released from the presynaptic membrane, promoting the transmission of neuromuscular excitability, enabling muscle contraction and movement. When the acetylcholine receptors are damaged, symptoms of muscle weakness occur. The most common causes of this acquired autoimmune disease are thymoma, thymic hyperplasia, or incomplete thymic involution, leading to the production of antibodies by the body that damage the acetylcholine receptors on the postsynaptic membrane. This damage results in a significant reduction of receptors, insufficient production of endplate potentials, and a disruption in transmission function, triggering myasthenia gravis.