Can rickets be cured?

Written by Li Jiao Yan
Neonatology
Updated on September 01, 2024
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Can rickets be cured? Rickets, also known as nutritional vitamin D deficiency rickets, is primarily caused by insufficient levels of vitamin D in the body leading to disorders in calcium and phosphorus metabolism. It is a chronic nutritional disease characterized by skeletal lesions. Typically, it manifests as flawed mineralization of long bones and bone tissue at the growth plates. Vitamin D deficiency rickets is a self-limiting disease that can generally be cured if children have sufficient outdoor activities and adequate exposure to sunlight. Adequate sunlight exposure and physiological doses of vitamin D can treat rickets. Therefore, it is usually recommended that children supplement with 400 units of vitamin D starting about two weeks after birth to prevent and treat rickets. However, if the child is in a later stage and shows obvious skeletal deformities, corrective treatment may be necessary. In severe cases of rickets where intracranial lesions occur, it might affect the child's vital signs and, in serious cases, could even lead to the child's death. Therefore, it is advised that children get plenty of sunlight, routinely supplement with vitamin D, and have regular pediatric check-ups to assess their condition.

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Written by Li Jiao Yan
Neonatology
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Rickets is caused by a deficiency of what element?

Rickets is a systemic chronic nutritional disease caused by vitamin D deficiency leading to disturbances in calcium and phosphorus metabolism, which results in abnormal bone development. The main cause is a lack of vitamin D, generally related to feeding methods, the addition of cod liver oil, living environment, outdoor activity time, repeated respiratory infections, calcium deficiency during pregnancy, and the season of birth. It is generally recommended to routinely supplement vitamin D and cod liver oil after birth, have regular follow-ups, engage in appropriate outdoor activities, and during pregnancy, it is important to supplement with adequate calcium and vitamin D. Additionally, attention should be paid to children who have repeated respiratory and gastrointestinal diseases, as these can also affect the absorption of vitamin D and calcium and phosphorus. Thus, it is vital to actively prevent other diseases that may affect the absorption of vitamin D and to treat illnesses proactively when they occur.

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Written by Li Jiao Yan
Neonatology
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Early symptoms of rickets

Rickets is a chronic nutritional disease primarily caused by a deficiency of Vitamin D, which leads to disturbances in calcium and phosphorus metabolism in the body, resulting in abnormal bone development. It is mostly seen in infants, especially common within the first six months, and particularly in babies younger than three months. Clinically, the symptoms are not very specific; the children may appear irritable and restless, they might cry suddenly, or become agitated and restless to the touch. Some children may exhibit excessive sweating, frequent head shaking, or even hair loss, leading to noticeable baldness at the back of the head. However, these symptoms are not very specific. Generally, blood biochemistry and Vitamin D levels need to be tested. Early X-ray findings of the bones can be normal.

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Written by Li Jiao Yan
Neonatology
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Rickets symptoms

Rickets, also known as nutritional vitamin D deficiency rickets, is a chronic nutritional disease in children caused by inadequate vitamin D in their bodies, leading to disorders in calcium and phosphorus metabolism and characterized by changes in the growth plates. Its manifestation varies with different ages. In infants under six months, especially those under three months, symptoms often include increased nervous excitability, such as being easily irritable, restless, sweating, and shaking their heads due to scalp irritation. As the condition progresses in children under six months, the primary changes are seen in the skull; the edges of the anterior fontanelle are softer, and the skull is thinner. After six months, the softening of the skull disappears, but there can be ping-pong ball-like changes around the skull, leading to a box-shaped head from seven to eight months. Gradually, beading changes form, most notably around the 7th to 10th ribs. Around the age of one, children can develop a pigeon chest deformity, and in severe cases of rickets, a horizontal depression forms at the lower edge of the thoracic cage, known as the costal groove or Harrison's groove. Due to bone softening and muscle and joint laxity, when the child begins to stand and walk, the legs may become bow-legged or X-shaped, and in severe cases, can develop into 'K'-shaped leg deformities. Once the child starts to sit and stand, general ligament laxity can cause spinal deformities. Severe hypophosphatemia leads to muscle carbohydrate metabolism disorder, resulting in overall muscle laxity, decreased muscle tone, and subsequently reduced muscle strength.

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Written by Li Jiao Yan
Neonatology
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Rickets is a disease that affects bone development in children due to vitamin D deficiency, calcium, or phosphate. It is treatable with proper nutrition and supplementation.

Rickets, also known as vitamin D deficiency rickets, is caused by a lack of vitamin D, leading to abnormal calcium and phosphorus metabolism in the body. The calcium salts cannot be properly deposited in the growing parts of the skeleton, causing incomplete mineralization of the bone tissue during growth. This results in a chronic systemic nutritional disease closely related to lifestyle, characterized by skeletal abnormalities. The most common is nutritional vitamin D deficiency, which is self-limiting. Typically, sufficient vitamin D supplementation and appropriate sun exposure can cure it. If caused by other severe diseases, vitamin D deficiency will significantly manifest liver and kidney function abnormalities. Additionally, some congenital genetic diseases make treatment more difficult when vitamin D levels are deficient.

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Written by Li Jiao Yan
Neonatology
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Early symptoms of rickets

Rickets is a chronic systemic nutritional disease caused by insufficient vitamin D in the body, leading to disturbances in calcium and phosphorus metabolism, characterized by skeletal lesions, typically presenting as incomplete mineralization of the long bones' growth plates and bone tissue. In the early stages of rickets, skeletal development abnormalities are not very obvious. Infants under three months often exhibit signs of increased neural excitability, such as being prone to crying, sweating easily, and potentially frequent head shaking; however, these symptoms are not necessarily specific to rickets. Generally, at this time, there are no obvious abnormalities in skeletal development, and X-rays of the bones are normal. The focus is on vitamin levels, specifically decreased serum 25-hydroxy vitamin D, as well as reduced blood calcium and phosphorus levels. Therefore, early symptoms are not so typical. It is recommended that children regularly visit pediatric clinics for physical examinations to assess their development and, if necessary, undergo trace element and vitamin D level checks.