Initial symptoms of cerebral embolism

Written by Liu Yan Hao
Neurology
Updated on December 29, 2024
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The initial characteristics of cerebral embolism generally include a sudden onset and rapid progression of the condition. It often occurs during physical activity, with the condition reaching a peak in a very short time. Typically, within a short period, this can lead to complete paralysis of limbs on one side, complete loss of speech in some cases, and severe cases may quickly progress into a coma. The development of the condition is quite rapid.

Common sources of emboli in cerebral embolism include mural thrombi from the heart and detached plaques from the carotid artery. Carotid artery plaques, especially soft plaques, are prone to detachment, forming emboli that block the cerebral vessels and cause cerebral embolism. Additionally, patients with arrhythmias or atrial fibrillation can easily form mural thrombi on the inner walls of the heart. Once a thrombus detaches, it can block cerebral vessels, leading to cerebral embolism. The affected area in cerebral embolism is relatively large, and the condition is quite severe.

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Written by Liu Yan Hao
Neurology
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How to treat cerebral embolism?

Brain embolism generally blocks relatively larger cerebral blood vessels and often leads to extensive death of brain cells. Therefore, patients with brain embolism are primarily treated with medications that promote blood circulation and remove blood stasis to improve circulation. Additionally, patients with brain embolism often experience an increase in intracranial pressure, so it is necessary to use medications that reduce intracranial pressure and alleviate brain cell edema. Patients with brain embolism might experience disturbances in consciousness and are prone to develop aspiration pneumonia or dependent pneumonia, so anti-infection medications may be utilized for treatment. Some may also suffer from stress-related gastrointestinal ulcers, or even gastrointestinal ulcer bleeding, thus requiring medications that inhibit gastric acid secretion and protect the gastric mucosa. Other treatments target the cause of the condition. A common cause is the detachment of carotid artery plaques, therefore, medications that prevent arteriosclerosis and stabilize plaques are also used. (Please use medications under the guidance of a doctor.)

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Written by Zhang Hui
Neurology
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Is cerebral embolism the same as cerebral infarction?

Brain embolism is a form of cerebral infarction, so essentially, it is also a type of cerebral infarction. Cerebral infarction encompasses a wider range, including cerebral thrombosis, lacunar infarction, watershed infarction, and others. Brain embolism primarily refers to abnormal substances entering the bloodstream, which then enter the arteries of the brain, causing obstruction in these arteries and leading to ischemia and hypoxia of the brain tissue, thus presenting clinical symptoms of cerebral infarction. The onset of the condition in patients is quite severe, rapidly leading to paralysis of limbs and disorders of speech function. In cases of extensive brain embolism, patients may even experience coma and death as serious complications. Most patients with brain embolism have a history of atrial fibrillation. Atrial fibrillation can lead to the formation of mural thrombi, and when these thrombi dislodge, they can cause brain embolism.

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Written by Liu Yan Hao
Neurology
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Can people with cerebral embolism run?

Patients with cerebral embolism can jog moderately, but walking is highly recommended. This is because the common source of the embolus in cerebral embolism patients is a mural thrombus in the heart. The common causes of mural thrombus formation include arrhythmia and atrial fibrillation. Therefore, patients with cerebral embolism, who may also have coronary artery disease, atrial fibrillation, or cardiac mural thrombus, are not well-suited for running, which is a relatively intense form of exercise. They can jog moderately, but the distance and duration should not be too long. The most recommended form of exercise is walking. Walking is the best form of exercise to prevent worsening the condition after running, and it could also exacerbate coronary artery disease conditions.

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Written by Zhang Hui
Neurology
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How many days for brain embolism infusion?

Cerebral embolism is a particularly dangerous disease that is very concentrated. It primarily refers to the entry of abnormal substances into the arteries, leading to the occlusion of the cerebral arteries and causing corresponding clinical manifestations in patients. Symptoms may include disturbances in consciousness, coma, limb paralysis, numbness, slurred speech, and even possibly seizures. The most common cause of cerebral embolism is atrial fibrillation. Treatment generally requires hospitalization for intravenous infusion. If the condition is relatively stable, an infusion lasting about 10 to 14 days may suffice, followed by administration of anticoagulant drugs for treatment. If the area affected by the cerebral embolism is very large and the patient is in severe danger, infusion treatment may need to last about 3 to 4 weeks. The specific approach should be based on the patient's condition. Additionally, certain medications to reduce dehydration and intracranial pressure, as well as neuroprotective drugs, should be administered. (Please use medication under the guidance of a doctor.)

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Written by Liu Yan Hao
Neurology
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Clinical symptoms of cerebral embolism

Patients with cerebral embolism typically exhibit a sudden onset and rapid progression of the condition, often occurring during physical activity and rapidly reaching its peak. Clinically, this can result in immediate and complete paralysis of one side of the body or total aphasia, and in some cases, it can also lead to a swift onset of coma. Additionally, these patients often experience accompanying conditions such as arrhythmias, carotid artery plaque formation, and arteriosclerosis. The common sources of emboli in cerebral embolism are thrombi attached to the heart wall or plaques formed in the carotid artery. Therefore, these patients often have a history of hyperlipidemia, arteriosclerosis, plaque formation in arteries, atrial fibrillation, or coronary artery disease.