Clinical symptoms of cerebral embolism

Written by Liu Yan Hao
Neurology
Updated on March 12, 2025
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Patients with cerebral embolism typically exhibit a sudden onset and rapid progression of the condition, often occurring during physical activity and rapidly reaching its peak. Clinically, this can result in immediate and complete paralysis of one side of the body or total aphasia, and in some cases, it can also lead to a swift onset of coma. Additionally, these patients often experience accompanying conditions such as arrhythmias, carotid artery plaque formation, and arteriosclerosis. The common sources of emboli in cerebral embolism are thrombi attached to the heart wall or plaques formed in the carotid artery. Therefore, these patients often have a history of hyperlipidemia, arteriosclerosis, plaque formation in arteries, atrial fibrillation, or coronary artery disease.

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Written by Liu Yan Hao
Neurology
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What are the symptoms of cerebral embolism?

Brain embolism usually begins suddenly and develops rapidly, often reaching a peak in a very short time. This can cause patients to quickly become comatose or experience weakness in limbs on one side of the body, with some presenting with complete paralysis or speech impairments. These symptoms occur because a major blood vessel in the brain is blocked abruptly, leading to ischemia and tissue necrosis in the supplied area. Common sources of the emboli include plaques in the cervical arteries, where soft plaques can dislodge and form emboli that block cerebral vessels with the bloodstream. Alternatively, patients with atrial fibrillation may suffer from embolism when thrombi attached to the inner walls of the heart dislodge and block cerebral vessels through circulation. Brain embolism is generally a severe condition with a rapid onset.

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Written by Zhang Hui
Neurology
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Symptoms and manifestations of cerebral embolism

The so-called cerebral embolism primarily refers to the presence of abnormal substances in the arteries, which block the smaller arteries leading to arterial occlusion, causing severe cerebral ischemia and hypoxia, and resulting in the necrosis of brain tissue. It generally occurs in patients with atrial fibrillation, where atrial fibrillation can form larger emboli that detach from the heart and can easily cause cerebral embolism. The symptoms of the onset are very sudden and rapidly reach their peak. Patients may experience coma and consciousness disorders, fixed staring of the eyes, paralysis of limbs, numbness of limbs, and an inability to sense pain and temperature in the limbs. Additionally, some may experience epileptic seizures and urinary and fecal incontinence. Cerebral embolism is a very dangerous disease, and once these symptoms appear, it is crucial to seek hospital treatment as soon as possible.

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Written by Zhang Hui
Neurology
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Does cerebral embolism require surgery?

Most cases of cerebral embolism in patients are caused by heart diseases, especially in patients with atrial fibrillation. Thrombi form in the atrium, detach, and travel to the cerebral arteries, causing a cerebral embolism. If the area affected by the cerebral embolism is small and the symptoms are mild, surgical treatment may not be necessary. Patients should rest, engage in rehabilitation exercises, and be treated with medications that invigorate the blood, remove blood stasis, protect brain cells, and use anticoagulants. However, if the embolism affects a large area, such as the middle cerebral artery, interventional surgical thrombectomy can be performed according to current medical advancements, which may be effective for some patients but also carries risks of bleeding and potential surgical failure. (Please use medications under the guidance of a doctor and do not self-medicate.)

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Written by Zhang Hui
Neurology
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What to do about cerebral embolism?

The onset of cerebral embolism is very sudden, and most cases of cerebral embolism are caused by heart disease. For instance, atrial fibrillation or myocardial infarction can lead to cerebral embolism, which requires immediate medical attention. If it is within the time window for arterial thrombectomy, it is best to proceed with the arterial thrombectomy treatment, and this window typically ranges from six to eight hours. If the time window is missed, the patient should primarily be kept on bed rest and given medications to protect and nourish brain cells. If intracranial pressure is high, medications to reduce dehydration and lower cranial pressure should be administered. After the acute phase, anticoagulation treatment should also be provided to prevent future occurrences of cerebral embolism. Additionally, care should be taken to prevent complications such as pulmonary infections, urinary system infections, and bedsores.

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Written by Zhang Hui
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Is cerebral embolism the same as cerebral infarction?

Brain embolism is a form of cerebral infarction, so essentially, it is also a type of cerebral infarction. Cerebral infarction encompasses a wider range, including cerebral thrombosis, lacunar infarction, watershed infarction, and others. Brain embolism primarily refers to abnormal substances entering the bloodstream, which then enter the arteries of the brain, causing obstruction in these arteries and leading to ischemia and hypoxia of the brain tissue, thus presenting clinical symptoms of cerebral infarction. The onset of the condition in patients is quite severe, rapidly leading to paralysis of limbs and disorders of speech function. In cases of extensive brain embolism, patients may even experience coma and death as serious complications. Most patients with brain embolism have a history of atrial fibrillation. Atrial fibrillation can lead to the formation of mural thrombi, and when these thrombi dislodge, they can cause brain embolism.