symptoms of cerebral embolism

Written by Liu Yan Hao
Neurology
Updated on September 13, 2024
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Cerebral embolism generally has a rapid onset and develops quickly, often occurring during physical activity. Within a short period of time, the condition can peak, and patients may experience sudden coma or complete paralysis of one side of the body, as well as aphasia, dementia, and cognitive dysfunction. Overall, the progression of the disease is very fast and can reach a peak in a short time. These patients often have underlying diseases, such as atrial fibrillation, which is a type of arrhythmia. In patients with atrial fibrillation, mural thrombi can form on the inner walls of the heart. These thrombi can detach and, carried by the bloodstream, block cerebral vessels, leading to cerebral embolism. Additionally, some patients have a foundation of arteriosclerosis, particularly in the carotid arteries, which can form plaques, especially soft plaques. When these plaques detach, they can form emboli that block cerebral vessels, causing cerebral embolism.

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Written by Liu Yan Hao
Neurology
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Can people with cerebral embolism run?

Patients with cerebral embolism can jog moderately, but walking is highly recommended. This is because the common source of the embolus in cerebral embolism patients is a mural thrombus in the heart. The common causes of mural thrombus formation include arrhythmia and atrial fibrillation. Therefore, patients with cerebral embolism, who may also have coronary artery disease, atrial fibrillation, or cardiac mural thrombus, are not well-suited for running, which is a relatively intense form of exercise. They can jog moderately, but the distance and duration should not be too long. The most recommended form of exercise is walking. Walking is the best form of exercise to prevent worsening the condition after running, and it could also exacerbate coronary artery disease conditions.

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Written by Tang Bo
Neurology
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What should not be eaten in case of cerebral embolism?

Cerebral embolism is a type of cerebral infarction. It is a condition caused by the dislodgment of emboli, leading to the interruption of blood flow in the brain and resulting in the necrosis of brain tissue and cerebral infarction. There are no special dietary requirements for this condition, but it is generally advisable to follow a low-salt, low-fat diet, as high levels of salt or fats can be risk factors for cerebral infarction. Additionally, cerebral embolism can also be caused by atrial fibrillation. Patients with atrial fibrillation must monitor their coagulation function carefully and pay attention to cardiac issues. Regarding the diet, it should be low in salt and fat, and if the patient does not have diabetes, there is no need to pay special attention to blood sugar levels. However, if diabetes is present, diabetic dietary guidelines must be followed.

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Written by Liu Yan Hao
Neurology
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How to treat cerebral embolism?

Brain embolism generally blocks relatively larger cerebral blood vessels and often leads to extensive death of brain cells. Therefore, patients with brain embolism are primarily treated with medications that promote blood circulation and remove blood stasis to improve circulation. Additionally, patients with brain embolism often experience an increase in intracranial pressure, so it is necessary to use medications that reduce intracranial pressure and alleviate brain cell edema. Patients with brain embolism might experience disturbances in consciousness and are prone to develop aspiration pneumonia or dependent pneumonia, so anti-infection medications may be utilized for treatment. Some may also suffer from stress-related gastrointestinal ulcers, or even gastrointestinal ulcer bleeding, thus requiring medications that inhibit gastric acid secretion and protect the gastric mucosa. Other treatments target the cause of the condition. A common cause is the detachment of carotid artery plaques, therefore, medications that prevent arteriosclerosis and stabilize plaques are also used. (Please use medications under the guidance of a doctor.)

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Written by Liu Yan Hao
Neurology
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Clinical symptoms of cerebral embolism

Patients with cerebral embolism typically exhibit a sudden onset and rapid progression of the condition, often occurring during physical activity and rapidly reaching its peak. Clinically, this can result in immediate and complete paralysis of one side of the body or total aphasia, and in some cases, it can also lead to a swift onset of coma. Additionally, these patients often experience accompanying conditions such as arrhythmias, carotid artery plaque formation, and arteriosclerosis. The common sources of emboli in cerebral embolism are thrombi attached to the heart wall or plaques formed in the carotid artery. Therefore, these patients often have a history of hyperlipidemia, arteriosclerosis, plaque formation in arteries, atrial fibrillation, or coronary artery disease.

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Written by Zhang Hui
Neurology
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Which department should I go to for a cerebral embolism?

The patient has experienced a cerebral embolism, and it is definitely recommended to seek appropriate medical care in neurology. Neurologists have seen many patients with cerebral embolism and have rich experience in the pathogenesis, diagnosis, and treatment of this disease. The onset of cerebral embolism is quite severe, and patients generally may have a history of cardiac diseases, such as atrial fibrillation. It is crucial to go to the hospital as soon as possible if a cerebral embolism occurs. If within the time window for thrombectomy, such as within eight hours, relevant thrombectomy treatment can be performed. Additionally, some hospitals carry out thrombectomy treatments in neurosurgery or interventional departments. Therefore, if a cerebral embolism requires surgical treatment, one can also visit the interventional department or neurosurgery. After the condition of a cerebral embolism patient stabilizes, routine anticoagulation therapy is necessary to prevent a recurrence of the cerebral embolism.