Is it good to drink milk after a stroke?

Written by Gao Yi Shen
Neurosurgery
Updated on September 06, 2024
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Patients with cerebral infarction can drink milk, but it must be analyzed according to the specific condition at the time because patients with cerebral infarction often experience coma and accompanying dysphagia with coughing. In such cases, drinking milk is not a problem, but attention must be paid to changes in dietary habits. If necessary, a gastric tube must be inserted through the nose into the stomach for feeding to avoid direct oral intake, which can cause coughing, aspiration, and lead to pneumonia. Therefore, this point must be carefully considered. For ordinary people in daily life, a cup of milk a day is a very good choice because milk mainly contains protein and does not contain a lot of fat and sugar. For patients with cerebral infarction, this diet is very ideal. If fresh milk can be used in everyday life, it is more beneficial for promoting the patient's recovery.

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Is a lacunar infarct a minor stroke?

Lacunar stroke is, in a sense, a milder form of stroke. It may not involve complete paralysis of limbs or consciousness disorders. Such cases typically show symptoms of sudden large vessel blockage. However, it results from blockages in many small vessels and manifests in various ways. Additionally, it gradually worsens, displaying an increasing range of symptoms including cognitive impairments, dementia, unstable gait, slurred speech, and dysarthria. These symptoms significantly affect the quality of life, so it can be considered not mild.

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Causes of Cerebral Infarction

At present, there is no consensus on the specific causes of cerebral infarction. It is mostly seen in patients with underlying diseases such as hypertension, diabetes, and hyperlipidemia, where there has not been adequate control of the patient's blood pressure, blood sugar, and cholesterol. This can lead to significant increases in blood pressure, blood sugar, and cholesterol. Over time, this might result in the formation of atherosclerotic plaques in the patient’s blood vessels. When these plaques break off and enter the bloodstream, they travel with the blood flow and can lodge in the narrow vessels of the brain. This causes blockage of the blood vessel and leads to ischemia, necrosis, softening, and degeneration of the brain tissue in the affected blood supply area, ultimately causing a cerebral infarction.

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Can the blood vessels in a cerebral infarction be unblocked?

Whether the blood vessels in a cerebral infarction can be reopened is closely related to time. If it is within the hyperacute phase, which means within six hours from the onset of clinical symptoms to hospital admission, the majority of cases can undergo thrombolysis intravenously, or arterial thrombectomy to reopen the blood vessels. However, if the time exceeds this window, reopening is no longer feasible, and forced reopening at this stage can easily lead to the detachment of distal thrombi or cause reperfusion injury, which is more detrimental to the patient's condition. Therefore, it is impossible to reopen the blood vessels during the chronic phase or non-hyperacute phase. If treatment of this vascular occlusion is desired, vascular anastomosis can be considered, though the specific type of anastomosis surgery depends on the patient's specific condition at the time. Thus, the possibility of reopening a blood vessel in a cerebral infarction certainly depends on timing.

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Stroke treatment methods

For patients who suffer a stroke, it is crucial to immediately transport them to the nearest local hospital. At the emergency room of the local hospital, a cranial CT scan or MRI should be conducted to confirm the presence of a stroke, as well as to determine its exact range and severity. If there is an acute indication for surgery, thrombolytic therapy is recommended. If the optimal time window for thrombolysis has passed, it is advisable to use medications that invigorate the brain, improve blood flow to remove stasis, nourish the nerves, and enhance microcirculation in the brain tissue. This can effectively help improve the conditions of ischemia and hypoxia in the brain nerves. Furthermore, further treatment should be adjusted based on the patient’s condition. During treatment, attention should be paid to the prevention and treatment of various complications or comorbidities, such as pulmonary infections, urinary tract infections, hypoproteinemia, deep vein thrombosis, etc.

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How is a cerebral infarction treated?

The treatment principles for cerebral infarction include thrombolysis, anticoagulation, volume expansion, lowering blood pressure, and brain protection. The main goal is to improve the blood circulation in the ischemic area of the brain as soon as possible and promote the recovery of neurological functions. During the acute phase, patients should rest in bed as much as possible, enhance care for skin, oral cavity, respiratory tract, and excretion, and maintain electrolyte balance. If a patient still cannot eat 48 to 72 hours after onset, nasogastric feeding with liquid nutrition should be provided to ensure nutritional supply. The priority should be given to the patient's daily care, diet, and the management of other comorbidities. Since some patients with cerebral infarction cannot take care of themselves during the acute phase and may even have difficulty swallowing, without adequate nutrition, metabolic issues can arise quickly. In such cases, even the best medications can fail to achieve positive outcomes. Since cerebral thrombosis is the most common type of cerebral infarction characterized by high incidence, high mortality, high recurrence rate, and high disability rate, patients may experience mild hemiplegia or severe scenarios leading to loss of life. Therefore, lifelong medication is necessary to prevent recurrence, achieve secondary prevention, and ultimately reduce the recurrence of cerebral infarction.