Causes of Cerebral Infarction

Written by Chen Yu Fei
Neurosurgery
Updated on September 25, 2024
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At present, there is no consensus on the specific causes of cerebral infarction. It is mostly seen in patients with underlying diseases such as hypertension, diabetes, and hyperlipidemia, where there has not been adequate control of the patient's blood pressure, blood sugar, and cholesterol. This can lead to significant increases in blood pressure, blood sugar, and cholesterol. Over time, this might result in the formation of atherosclerotic plaques in the patient’s blood vessels. When these plaques break off and enter the bloodstream, they travel with the blood flow and can lodge in the narrow vessels of the brain. This causes blockage of the blood vessel and leads to ischemia, necrosis, softening, and degeneration of the brain tissue in the affected blood supply area, ultimately causing a cerebral infarction.

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Written by Chen Yu Fei
Neurosurgery
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Massage methods for cerebral infarction

For patients with cerebral infarction, many of them suffer from facial paralysis, often characterized by crooked mouth and eyes and drooling. To alleviate symptoms and help improve the asymmetry of the mouth and eyes, it is suggested to massage the facial and neck muscles of the patient. It is generally recommended to seek an experienced Traditional Chinese Medicine practitioner who can use manual massage techniques, targeting specific acupoints to effectively relieve pain and improve symptoms. For patients with motor dysfunction or even hemiplegia, manual massage can effectively help prevent muscle atrophy, relieve muscle rigidity, and reduce muscle tone.

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Written by Shu Zhi Qiang
Neurosurgery
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post-stroke sequelae

Brain infarction is a very common type of ischemic cerebrovascular disease, which mainly includes the following types: the most common are lacunar infarction, cerebral thrombosis, and cerebral embolism. The neurological sequelae of lacunar infarction are not particularly typical, and patients may exhibit mild incomplete paralysis of limbs, decreased muscle strength, reduced sensation, or ataxia. Cerebral thrombosis and cerebral embolism often cause extensive brain infarction, where patients typically experience severe limb paralysis, loss of sensation, increased tendon reflexes, positive pathological reflexes, and an inability to care for themselves.

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Written by Gao Yi Shen
Neurosurgery
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Premonitory symptoms of cerebral infarction recurrence

The symptoms preceding the recurrence of a cerebral infarction mainly involve observing the symptoms presented during the initial incidence of the stroke. In many cases, the recurrent symptoms are very similar to those initially experienced. For example, if dizziness was a symptom during the previous episode, then dizziness could also be a precursor symptom in a recurrence, with similar nature and intensity of dizziness as before. Additionally, if previous symptoms included limb numbness, weakness, or speech impairment, similar signs might precede a recurrence. Clinically, it is imperative to address these issues before an actual recurrence occurs, such as actively pursuing antiplatelet aggregation therapy, lipid-lowering, and stabilizing plaques, and resorting to surgical treatment if necessary, to better reduce the chances of stroke recurrence.

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Written by Gao Yi Shen
Neurosurgery
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What is a cerebral infarction?

Cerebral infarction is essentially a manifestation of ischemic necrosis of nerve cells. However, in clinical practice, there are many factors that can cause cerebral infarction. It is essential to treat the underlying causes to effectively prevent the further occurrence and progression of cerebral infarction. One characteristic of nerve cells is that once necrosis occurs, they cannot regenerate. Consequently, all patients who suffer from cerebral infarction may have some residual neurological deficits. Mild symptoms include headaches and dizziness, while severe cases may experience numbness and weakness in the limbs, speech impairments, and even more severe conditions such as a vegetative state or death. Therefore, in the case of cerebral infarction, it is crucial to focus on prevention early on to reduce the extent of necrosis. This can effectively ensure one's safety and reduce the occurrence of various complications and sequelae.

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Written by Gao Yi Shen
Neurosurgery
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Can the blood vessels in a cerebral infarction be unblocked?

Whether the blood vessels in a cerebral infarction can be reopened is closely related to time. If it is within the hyperacute phase, which means within six hours from the onset of clinical symptoms to hospital admission, the majority of cases can undergo thrombolysis intravenously, or arterial thrombectomy to reopen the blood vessels. However, if the time exceeds this window, reopening is no longer feasible, and forced reopening at this stage can easily lead to the detachment of distal thrombi or cause reperfusion injury, which is more detrimental to the patient's condition. Therefore, it is impossible to reopen the blood vessels during the chronic phase or non-hyperacute phase. If treatment of this vascular occlusion is desired, vascular anastomosis can be considered, though the specific type of anastomosis surgery depends on the patient's specific condition at the time. Thus, the possibility of reopening a blood vessel in a cerebral infarction certainly depends on timing.