What is a cerebral infarction?

Written by Gao Yi Shen
Neurosurgery
Updated on September 08, 2024
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Cerebral infarction is essentially a manifestation of ischemic necrosis of nerve cells. However, in clinical practice, there are many factors that can cause cerebral infarction. It is essential to treat the underlying causes to effectively prevent the further occurrence and progression of cerebral infarction. One characteristic of nerve cells is that once necrosis occurs, they cannot regenerate. Consequently, all patients who suffer from cerebral infarction may have some residual neurological deficits. Mild symptoms include headaches and dizziness, while severe cases may experience numbness and weakness in the limbs, speech impairments, and even more severe conditions such as a vegetative state or death. Therefore, in the case of cerebral infarction, it is crucial to focus on prevention early on to reduce the extent of necrosis. This can effectively ensure one's safety and reduce the occurrence of various complications and sequelae.

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Written by Gao Yi Shen
Neurosurgery
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Is a grade II cerebral infarction serious?

A second-degree cerebral infarction is a very serious matter that must be given significant attention clinically. Once a cerebral infarction occurs, it indicates very poor vascular quality. If proactive treatment and prevention are not undertaken to prevent further progression, the symptoms of cerebral infarction can worsen and recur frequently. This can cause severe harm not only to the patient but also to their family and society. This is particularly true for patients with severe, long-term bedridden hemiplegia, which further increases the burden on families and society. Therefore, once a cerebral infarction occurs, regardless of the circumstances, it is crucial to complete the necessary examinations and take timely treatment measures to better manage the progression of the condition.

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Written by Liu Dong Yang
Neurosurgery
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Can a cerebral infarction recover?

Most patients who experience a stroke have relatively good recovery after active treatment and rehabilitation exercise, but very few patients can recover to the physical state they were in before the stroke. This is mainly because after the stroke, brain cells are damaged due to ischemia and hypoxia. Since brain cells are non-regenerative tissues, it is difficult for them to regenerate once they are damaged. The function of the damaged brain cells is mainly compensated by the undamaged brain neurons. Therefore, even with active treatment, it's not possible to return to a completely normal state. However, even so, it is still crucial to actively pursue treatment and rehabilitation exercises after a stroke, as these can significantly reduce the disability and mortality rates caused by the stroke.

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Written by Gao Yi Shen
Neurosurgery
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Methods of Cerebral Infarction

There are many methods for treating cerebral infarction, but it is essential to make judgments based on the situation, as each patient's condition is different and requires different treatment approaches. For patients with acute cerebral infarction, treatments such as emergency thrombolysis or thrombectomy can be undertaken. For chronic cerebral infarction, initially, medical treatment is applied, including managing blood pressure, lowering blood lipids, stabilizing blood sugar levels, quitting smoking and alcohol, etc. Subsequently, this includes the oral administration of antiplatelet aggregation medications, which can effectively reduce the recurrence of cerebral infarction. Finally, corresponding surgical treatments are considered, including endarterectomy, superficial temporal artery to middle cerebral artery anastomosis, and appropriate stent implantation surgeries, depending on the specific characteristics. (Specific medications should be administered under the guidance of a physician.)

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Written by Shu Zhi Qiang
Neurosurgery
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post-stroke sequelae

Brain infarction is a very common type of ischemic cerebrovascular disease, which mainly includes the following types: the most common are lacunar infarction, cerebral thrombosis, and cerebral embolism. The neurological sequelae of lacunar infarction are not particularly typical, and patients may exhibit mild incomplete paralysis of limbs, decreased muscle strength, reduced sensation, or ataxia. Cerebral thrombosis and cerebral embolism often cause extensive brain infarction, where patients typically experience severe limb paralysis, loss of sensation, increased tendon reflexes, positive pathological reflexes, and an inability to care for themselves.

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Written by Gao Yi Shen
Neurosurgery
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Can the blood vessels in a cerebral infarction be unblocked?

Whether the blood vessels in a cerebral infarction can be reopened is closely related to time. If it is within the hyperacute phase, which means within six hours from the onset of clinical symptoms to hospital admission, the majority of cases can undergo thrombolysis intravenously, or arterial thrombectomy to reopen the blood vessels. However, if the time exceeds this window, reopening is no longer feasible, and forced reopening at this stage can easily lead to the detachment of distal thrombi or cause reperfusion injury, which is more detrimental to the patient's condition. Therefore, it is impossible to reopen the blood vessels during the chronic phase or non-hyperacute phase. If treatment of this vascular occlusion is desired, vascular anastomosis can be considered, though the specific type of anastomosis surgery depends on the patient's specific condition at the time. Thus, the possibility of reopening a blood vessel in a cerebral infarction certainly depends on timing.