Causes of hoarseness due to ventricular septal defect.

Written by Di Zhi Yong

Cardiology

Updated on September 07, 2024

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If a patient exhibits some ventricular septal defects, it is possible for hoarseness to occur, as this condition is a type of congenital heart disease.

If a patient frequently experiences hoarseness, it might be caused by congenital heart disease, but chronic pharyngitis or laryngitis, which can also cause hoarseness, should not be ruled out. Additionally, conditions like vocal cord polyps or deeper diseases can lead to hoarseness. However, it is important to differentiate these from normal diseases. If the issue is solely a ventricular septal defect, surgical treatment can be recommended for the patient, which can achieve a complete cure. Attention should also be paid to maintaining a healthy diet.

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Written by Chen Tian Hua

Cardiology

56sec

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Ventricular septal defect is what?

Ventricular septal defect is a common congenital heart disease, mainly due to the abnormal defect in the ventricular septum during the fetal development, resulting in an abnormal channel between the left and right ventricles. Ventricular septal defect can cause blood to shunt from the left ventricle to the right ventricle, gradually increasing the blood flow in the pulmonary circulation, leading to an increase in the volume load of the left and right ventricles, while causing a decrease in the systemic circulation volume. In the later stages, due to the significant increase in right ventricular pressure, it leads to right-to-left shunting, thereby causing Eisenmenger's syndrome. For ventricular septal defects with a large degree of defect, timely interventional treatment or surgical ventricular septal defect repair is required.

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Written by Xie Zhi Hong

Cardiology

1min

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Does interventricular septal defect occasionally cause chest pain?

Ventricular septal defect is a type of congenital heart disease, occurring when the ventricles of the heart develop poorly during the embryonic stage, leading to a defect in the ventricular septum. This often results in a left-to-right shunt which affects the efficiency of the heart's function. In severe cases, it can cause a right-to-left shunt. Generally, these conditions do not affect the coronary circulation or cause chest pain in patients. However, some patients may develop endocarditis at the ventricular septum, which sometimes leads to the formation of vegetations on the cardiac valves or blood clots on the abdominal wall. If thrombosis or vegetations occur, there is a possibility of these breaking off. If they enter the coronary arteries, they can cause chest pain. However, such events are extremely rare, with an occurrence rate of less than one in a thousand.

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Written by Xie Zhi Hong

Cardiology

1min 42sec

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Is perimembranous ventricular septal defect serious?

The ventricles are separated by the interventricular septum, which divides the left and right ventricles. Typically, after blood is ejected from the left ventricle and oxygenated by tissues, it returns to the right ventricle. The blood from the right ventricle gets oxygenated through the pulmonary artery before it can return to the left ventricle. These two should be isolated from each other before passing through the lungs. The interventricular septum consists of the membranous and muscular parts, and a defect in either part can affect oxygenation. Therefore, whether the defect is in the membranous or muscular portion is not an indicator for diagnosing the severity of the interventricular septum defect; rather, the size of the defect should be considered. Generally speaking, a defect of about 10mm in the interventricular septum, especially when accompanied by other conditions such as Tetralogy of Fallot, is considered severe. Moreover, when the defect reaches over 10mm, surgical intervention is required as minimally invasive surgery would not be feasible; these are considered severe interventricular septum defects. Also, there are cases with multiple complex defects or larger defects that should be surgically treated as soon as possible.

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Written by Xie Zhi Hong

Cardiology

1min 14sec

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Will being overweight affect the surgery for a ventricular septal defect?

Ventricular septal defect surgery is performed by puncturing the right femoral vein, guiding the catheter into the inferior vena cava, through the atria and ventricles, and reaching the defect via the right ventricle. The occluder can then be delivered through the catheter into the left ventricle, where the umbrella is opened, followed by opening it in the right ventricle; this describes the surgical process. Therefore, generally speaking, the surgical process is not affected in most patients who are obese. However, severe obesity can impact the accuracy of vascular localization during puncture by the surgeon. Secondly, if complications such as anesthesia issues or other emergencies arise during the surgery, severely obese patients may experience a reduced success rate in emergency surgical interventions, hence it is recommended that patients control their weight. Furthermore, severe obesity can exacerbate the cardiac load originally induced by the ventricular septal defect, thus affecting the surgical tolerance in patients whose cardiac function is already compromised by the defect. It is recommended that these patients control their weight.

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Written by Xie Zhi Hong

Cardiology

45sec

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What should I do about hypoxia due to a ventricular septal defect?

Mild ventricular septal defects generally do not cause hypoxia. If symptoms of hypoxia recur due to a ventricular septal defect, it is recommended to undergo minimally invasive interventional treatment or surgical treatment at a hospital to prevent further episodes. The success rate of this surgery is very high, almost 100%, with relatively few complications. If hypoxia occurs in untreated ventricular septal defects, initial therapy should include oxygen administration, followed by diuretic, cardiotonic, and other treatments to reduce cardiac load at a hospital, and surgery should be considered as soon as suitable.