Is acute heart failure dangerous?

Written by Wang Li Bing
Intensive Care Medicine Department
Updated on September 20, 2024
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Acute heart failure is very dangerous in clinical practice. It is mainly caused by a sudden decrease in cardiac output, leading to insufficient perfusion in tissues and organs and acute blood stasis syndrome. The main clinical symptoms include severe respiratory distress, often accompanied by restlessness, profuse sweating, frequent coughing with pink frothy sputum, and in severe cases, sudden cardiac and respiratory arrest can occur. After the occurrence of acute heart failure, effective measures should be taken to alleviate symptoms such as respiratory distress. The most effective methods in clinical practice include strengthening the heart, promoting diuresis, and dilating blood vessels.

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Written by Wang Li Bing
Intensive Care Medicine Department
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The basic mechanisms of heart failure

Heart failure is not an independent disease; it can be caused by various factors, such as myocardial infarction, cardiomyopathy, and various myocarditis, which lead to changes in the heart structure and subsequent cardiac dysfunction. Heart failure is mainly due to a disturbance in the heart's contractile function or diastolic function, which prevents the heart from adequately ejecting the returning blood volume. This results in pulmonary congestion, systemic circulation congestion, and a series of syndromes caused by insufficient arterial perfusion.

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Written by Zhang Yue Mei
Cardiology
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Heart Failure Drug Therapy

Due to some myocardial diseases that cause damage to the myocardium, or other reasons that increase the heart's load, heart failure may occur. Clinical treatment involves strengthening the heart and promoting diuresis, improving blood vessel function, increasing the heart’s contractility, reducing the heart’s resistance, and decreasing the volume of blood returning to the heart. Through these effective drug treatments, heart failure can be corrected. Patients with heart failure should avoid excessive fatigue and maintain a low-salt diet to prevent increasing the burden on the heart.

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Written by Wang Li Bing
Intensive Care Medicine Department
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Causes of vomiting in heart failure

Heart failure in clinical practice can be divided into left heart failure, right heart failure, and total heart failure. The symptoms of left heart failure primarily manifest as pulmonary congestion, including varying degrees of dyspnea, cough, coughing up pink frothy sputum, palpitations, fatigue, etc. As for right heart failure, it mainly shows as gastrointestinal congestion, with symptoms such as nausea, vomiting, loss of appetite, etc. Once heart failure symptoms appear, the patient must seek medical attention promptly, actively treat the primary disease, correct the symptoms and signs of heart failure, and then avoid excessive physical activity, infections, and other triggers that could exacerbate heart failure.

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Written by Wang Li Bing
Intensive Care Medicine Department
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Why does heart failure cause ascites?

Heart failure is relatively common in clinical settings, primarily divided into left heart failure, right heart failure, and global heart failure. The clinical manifestations of left heart failure are mainly reflected in symptoms of pulmonary congestion. It can present as varying degrees of dyspnea, cough, expectoration, palpitations, and fatigue. Right heart failure is mainly due to obstruction of the inferior vena cava flow, leading to increased venous pressure and capillary pressure. This causes plasma components to extravasate and ultimately leads to the formation of ascites. Global heart failure includes the symptoms of both left and right heart failure. Therefore, it is essential to seek medical attention promptly after heart failure occurs.

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Written by Wang Li Bing
Intensive Care Medicine Department
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How is acute heart failure treated?

Acute heart failure presents with significant respiratory distress and symptoms of hypoxia. Immediate intervention is necessary to alleviate breathing difficulties. Clinically, the following measures are generally adopted: First, position the patient to sit up with legs dangling to decrease venous return to the heart. Second, administer high-flow oxygen through a nasal cannula, and if necessary, proceed with endotracheal intubation and ventilator support. Third, administer morphine subcutaneously or intravenously to reduce agitation and myocardial oxygen demand. Fourth, use rapid diuretics, such as furosemide. Fifth, utilize vasodilators such as nitroglycerin or nitroprusside. Sixth, use positive inotropic agents such as dopamine or dobutamine as appropriate. Seventh, employ digitalis drugs, but avoid use in acute myocardial infarction within the first 24 hours. Eighth, consider using an intra-aortic balloon pump or other cardiopulmonary support systems, etc., and after stabilization of acute heart failure, further treat the underlying cause, etc. (Use medications under the guidance of a doctor.)