

Zhang Hui

About me
Weifang People's Hospital, Department of Neurology, attending physician, has been engaged in clinical work in the field of neurology for many years, with rich clinical experience in common and prevalent neurological diseases.
Proficient in diseases
Cerebrovascular disease, Parkinson's disease, myelitis, etc.

Voices

MRI manifestations of brain atrophy
Patients with cerebral atrophy display clear signs on cranial Magnetic Resonance Imaging (MRI). The manifestations on MRI primarily include notably widened cerebral sulci, obvious atrophy of the cerebral lobes, and enlarged ventricles, which are typical presentations of cerebral atrophy. Cerebral atrophy can be categorized into atrophy of the cerebrum, cerebellum, and brainstem, each showing different characteristics on MRI, which requires careful differentiation. There are many causes of cerebral atrophy, such as degenerative diseases like Alzheimer's disease, which leads to cerebral atrophy. Cerebral thrombosis and poor cerebral vascular conditions can also cause cerebral atrophy due to insufficient blood supply to the brain. Additionally, drug toxicity and chronic alcoholism can lead to cerebral atrophy, which also needs to be differentiated carefully.

Alzheimer's disease is also called dementia.
Alzheimer's disease, also known as dementia in the elderly, was first identified abroad and thus named after the discoverer, Alzheimer. It is one of the most common neurodegenerative diseases in neurology, with a very high incidence among people over 65 in China and worldwide. The disease mainly affects patients' cognitive functions. Initially, patients do not meet the criteria for dementia and primarily exhibit a decline in memory. As the disease progresses, the decline in memory worsens, and patients begin to forget both recent and past events. Additionally, they may experience impairments in visuospatial abilities, judgment, comprehension, and learning capabilities. In the later stages, psychiatric symptoms and personality changes may appear, such as visual hallucinations, shouting, and gesturing wildly, among various other clinical manifestations.

Can cerebellar atrophy be cured?
Cerebellar atrophy is difficult to cure. There are many causes of cerebellar atrophy, such as degenerative diseases of the nervous system. For example, olivopontocerebellar atrophy, which is caused by a disease with an unknown pathogenesis leading to the death of cerebellar cells and resulting in atrophy, currently lacks effective treatment methods and is incurable. Some cases are due to hereditary diseases, such as spinocerebellar ataxia, where patients also suffer from cerebellar atrophy, making it very difficult to cure. In addition, some patients who have been chronically drinking alcohol can also develop cerebellar atrophy due to alcohol poisoning. Although clinical symptoms can be significantly improved by discontinuing alcohol use and administering large amounts of vitamin B1 and B12, the atrophy in the cerebellum cannot be reversed as seen on imaging studies.

How to alleviate myasthenia gravis?
Myasthenia gravis is a disease of the department of neurology and its incidence is not low, it is also relatively common clinically. The treatment of myasthenia gravis mainly includes the following points: First, if patients with myasthenia gravis also have a thymoma, it is recommended to surgically remove the thymoma as soon as possible, which can significantly alleviate the symptoms of myasthenia gravis and reduce the amount of medication needed. Second, provide patients with some medication, mainly some drugs that suppress the immune response, including some corticosteroids. The use of corticosteroids is relatively complex, and must be gradually increased or decreased under the advice of a doctor. Do not stop taking them on your own. There are also some drugs that are cholinesterase inhibitors, which can increase the content of acetylcholine in the synaptic gap, alleviating the patient's symptoms. In addition, research has confirmed that some new immunosuppressive agents can also be used in the treatment of myasthenia gravis. In summary, the treatment of myasthenia gravis is a comprehensive subject and must be tailored according to the patient's condition.

Can optic neuritis be treated without hospitalization?
Patients with optic neuritis generally experience severe symptoms, as failing to receive appropriate treatment can lead to a significant and potentially irreversible decline in vision, resulting in disability. It is recommended that patients with optic neuritis be hospitalized for treatment. Hospitalization is necessary to carry out various supportive tests, including lumbar puncture lab tests and comprehensive MRI scans. Treatment of optic neuritis also involves administering high doses of corticosteroids based on the patient's condition. It is difficult for patients to manage such high doses of corticosteroids at home. Moreover, doctors need to closely monitor for any adverse reactions caused by these steroids. Therefore, hospitalization is advised for the treatment of optic neuritis, as the risks and prognosis are considerably worse without it.

Which department should I go to for a cerebral embolism?
The patient has experienced a cerebral embolism, and it is definitely recommended to seek appropriate medical care in neurology. Neurologists have seen many patients with cerebral embolism and have rich experience in the pathogenesis, diagnosis, and treatment of this disease. The onset of cerebral embolism is quite severe, and patients generally may have a history of cardiac diseases, such as atrial fibrillation. It is crucial to go to the hospital as soon as possible if a cerebral embolism occurs. If within the time window for thrombectomy, such as within eight hours, relevant thrombectomy treatment can be performed. Additionally, some hospitals carry out thrombectomy treatments in neurosurgery or interventional departments. Therefore, if a cerebral embolism requires surgical treatment, one can also visit the interventional department or neurosurgery. After the condition of a cerebral embolism patient stabilizes, routine anticoagulation therapy is necessary to prevent a recurrence of the cerebral embolism.

Is it dangerous to have a fever with myasthenia gravis?
Myasthenia gravis is a disease of the neuromuscular junction, essentially an autoimmune inflammatory response that causes generalized weakness and fatigue of the skeletal muscles, with symptoms that are less severe in the morning and worsen by the evening, and fluctuate over time. The weakness can be alleviated by rest. When patients with myasthenia gravis develop a fever, it is generally considered to be caused by an infection, perhaps a viral infection such as an upper respiratory tract infection, which can also induce fever. In such cases, the main treatment involves drinking plenty of water and consuming fresh vegetables and fruits. If necessary, some antiviral medications and antipyretics may be administered. If the fever is due to a bacterial infection, symptomatic treatment should be accompanied by the appropriate antibiotics. However, the choice of antibiotics must be made with caution. Patients with myasthenia gravis should not use aminoglycosides or fluoroquinolones, as these could exacerbate their condition. Penicillins or cephalosporins may be used instead. (Please consult a professional physician for specific medication guidance.)

Is brain atrophy hereditary?
There are many causes of brain atrophy; whether it is hereditary depends on the specific disease causing it. Most diseases leading to brain atrophy are not hereditary. For instance, some patients suffer brain atrophy due to Alzheimer's disease, where the neurons die irreversibly. This disease is mostly sporadic and does not have a clear hereditary tendency. Of course, a few cases of Alzheimer's do have a family history and may have a genetic propensity. Additionally, brain atrophy can also occur due to intoxication, such as from alcohol, carbon monoxide, or other toxic substances, and these cases definitely are not hereditary. Some patients experience brain atrophy due to cerebral thrombosis, which also lacks a genetic predisposition.

Can people with optic neuritis eat spicy food?
Optic neuritis is essentially an inflammatory response of the immune system, primarily caused by disorders of the body's own immune functions. Patients with this condition are advised not to consume spicy foods, as these can lead to more pronounced immune dysfunction and potentially trigger these degenerative diseases. Therefore, it is recommended to avoid spicy foods. Additionally, patients with optic neuritis often need to take corticosteroids or immunosuppressants long-term, and should not consume spicy foods while on these medications. Besides avoiding spicy foods, patients with optic neuritis should also focus on eating fresh vegetables and fruits to enhance resistance and adjust immune dysfunction. Furthermore, they should consume foods high in B vitamins, such as soybeans, millet, corn, and sorghum.

What should one eat for optic neuritis?
Optic neuritis is generally caused by the demyelination of the optic nerve, presenting as a clinical syndrome. Patients mainly exhibit significant vision loss, which can affect one or both eyes. It is crucial for those suffering from optic neuritis to receive timely treatment, including the consumption of group B vitamins and corticosteroid medications. Dietary considerations are also important: first, patients should consume foods rich in B vitamins, which include lean meats, fish, soybeans, buckwheat, corn, millet, and sorghum, all of which contain abundant B vitamins. Second, a higher intake of fresh vegetables and fruits is advised, as these contain plenty of folic acid and vitamins, which can aid in the recovery from optic neuritis. Third, the consumption of foods rich in vitamin B12, essential for the formation of myelin in the optic nerve, is also important.