Once intestinal intussusception occurs, only a small portion of small bowel intussusception can reduce spontaneously, becoming temporary small bowel intussusception, while intussusception involving the colon or repeated intussusception generally cannot reduce on its own. Due to the continuous spasm of the sheathed intestine, circulatory disturbances occur in the intussuscepted segment, initially impeding venous return, causing tissue congestion and edema, varicose veins, and mucous cells secreting large amounts of mucus into the intestinal lumen. This results in a jam-like gelatinous discharge mixed with blood and fecal matter. The bowel wall swells, worsening the obstruction of venous return, affecting the arteries, leading to insufficient blood supply, causing necrosis of the intestinal wall, and systemic toxicity symptoms. In severe cases, this can lead to intestinal perforation and peritonitis. Treatment generally involves air or barium enema or surgical methods.