Can babies with ventricular septal defect sleep on their stomachs?

Written by Di Zhi Yong
Cardiology
Updated on December 19, 2024
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If the child has an atrial septal defect, it is not recommended for them to sleep on their stomach because this position can increase the burden on the heart, leading to an overload of the heart.

This is a type of congenital heart disease. Currently, surgical treatment is primarily recommended, as early surgical intervention can alleviate the symptoms in children. Patients with ventricular septal defects typically display signs of ischemia and hypoxia, which can affect a child’s growth and development.

It is advised to periodically re-examine the heart through echocardiography. If the defect is relatively large, early surgical treatment is recommended. It is also important to actively prevent upper respiratory infections, which can be beneficial for the child. In terms of sleeping posture, it is not recommended to sleep on the stomach; the best position is lying on the left side, as this can help alleviate the burden on the child’s heart.

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Written by Tang Li
Cardiology
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Imaging manifestations of atrial septal defect

The imaging characteristics of ventricular septal defects are as follows: 1. Small defects: Normal cardiac silhouette, or left ventricular hypertrophy, with mild pulmonary congestion. 2. Medium defects: Left ventricular hypertrophy or biventricular hypertrophy, enlarged pulmonary artery segment, smaller aortic knob. 3. Large defects: Both ventricles enlarged, left atrium enlarged, prominent pulmonary artery segment, significant pulmonary congestion. When pulmonary hypertension with right-to-left shunting occurs, the pulmonary artery segment is markedly prominent, but the lung fields are clear, and the heart shadow is smaller than before.

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Written by Tang Li
Cardiology
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How does septal defect shunt?

Atrial septal defect is the most common congenital heart disease in adults, accounting for 20%-30% of all adult congenital heart conditions. The impact of an atrial septal defect on hemodynamics primarily depends on the amount of shunting. Due to higher pressure in the left atrium compared to the right atrium, a left-to-right shunt is formed. The amount of shunting depends on the size of the defect, as well as the compliance of the left and right ventricles and the relative resistance in the systemic and pulmonary circulations. Diseases that affect the compliance of the left ventricle, such as hypertension and coronary artery disease, can increase the extent of the left-to-right shunt. A continuous increase in pulmonary blood flow leads to pulmonary congestion, increasing the load on the right heart. Pulmonary vascular compliance decreases, progressing from functional pulmonary arterial hypertension to organic pulmonary arterial hypertension. As right heart system pressure continuously increases and eventually exceeds the pressure in the left heart system, the original left-to-right shunt reverses to become a right-to-left shunt, resulting in cyanosis.

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Written by Tang Li
Cardiology
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Do infant atrial septal defects heal by themselves?

Atrial septal defect is a common congenital heart disease, accounting for about 1/1500 of newborns, and is most commonly found in infants and children. The impact of atrial septal defects on hemodynamics mainly depends on the amount of shunting. Some studies confirm that atrial septal defects smaller than six millimeters can close on their own without external intervention. Therefore, when an atrial septal defect is discovered, it is necessary to regularly recheck the cardiac echocardiography to determine the size of the defect. If the atrial septal defect decreases in size as the patient ages, there is a possibility of self-healing. If the area of the patient's atrial septal defect does not change or gradually increases, regular echocardiography rechecks are needed, and cardiac catheterization may be performed if necessary.

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Written by Tang Li
Cardiology
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Can a ventricular septal defect heal itself?

Traditional views suggest that the best age to close atrial septal defects (ASDs) is between four and five years old, during preschool. The possibility of spontaneous closure of atrial septal defects larger than 8 millimeters in diameter is very low. If the defect is less than 4 millimeters, there is a possibility of healing. Currently, it is advocated that if an atrial septal defect still exists after the age of one year, and there is evident systolic murmur and fixed splitting of the second heart sound, or if heart catheterization and echocardiography show left-to-right shunting greater than 1.5:1, indicating a defect diameter of five to six millimeters or more, early surgery should be pursued to stop the left-to-right shunt to avoid causing pulmonary hypertension and endocarditis.

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Atrial septal defect can cause which complications?

Small atrial septal defects generally are asymptomatic, whereas medium to large atrial septal defects may result in pulmonary hypertension and right heart failure. The impact of an atrial septal defect on hemodynamics mainly depends on the amount of shunting, which in turn is determined by the size of the defect. It also relates to the compliance of the left and right ventricles and the relative resistance of the systemic and pulmonary circulation. Continued increase in pulmonary blood flow leads to pulmonary congestion, increasing right heart volume load. Pulmonary vascular compliance decreases, evolving from functional to organic pulmonary hypertension. This in turn continuously increases right heart system pressure until it surpasses the pressure of the left heart system, causing the original left-to-right shunt to reverse to a right-to-left shunt, leading to cyanosis. Atrial septal defects are generally asymptomatic but with the progression of the condition, symptoms such as exertional dyspnea, arrhythmias, and right heart failure may occur. In the advanced stages, about 15% of patients with severe pulmonary hypertension exhibit a right-to-left shunt and cyanosis, leading to the formation of Eisenmenger syndrome.