Arteriosclerosis is divided into several stages.

Written by Zeng Wei Jie
Cardiology
Updated on September 28, 2024
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Arterial atherosclerosis sequentially manifests as three major types: lipid spots and stripes, atheromatous and fibrous atheromatous plaques, and complex lesions. According to the process of its occurrence and development, it can be subdivided into six types. The first type is lipid spots, which are small yellow spots on the arterial intima that gradually develop into the second type, lipid stripes, where some yellow stripes appear on the arterial intima. Then there is the third type, pre-lesional plaque, where more lipid droplets appear extracellularly. The fourth type is the atheromatous plaque, where lipids accumulate excessively and form lipid pools, and the intimal structure will be damaged. The fifth type is the fibrous atheromatous plaque, which is the most characteristic lesion of arterial atherosclerosis. The sixth type is a complex lesion, representing a severe condition.

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Can arteriosclerosis be treated with moxibustion?

Arteriosclerosis can be treated with moxibustion. Moxibustion is a traditional disease treatment method in Chinese medicine, which can treat many diseases clinically. For patients with arteriosclerosis, moxibustion therapy can improve some clinical symptoms caused by arteriosclerosis. However, one should not rely solely on moxibustion for the treatment of atherosclerosis, as the mechanism of atherosclerosis is due to the disorder of lipid metabolism and the hardening of arterial walls caused by increased blood lipids. Treatment should include medication and dietary adjustments such as low-salt, low-sugar, and low-fat diets, and avoiding fatty meats, animal offal, and seafood high in cholesterol.

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How long can one live with arteriosclerosis?

Arteriosclerosis has a certain impact on survival and longevity, but it is not an absolute determinant. People with mild arteriosclerosis have fewer sudden incidents of cerebral infarction and myocardial infarction, thus they may have a longer survival period. However, if arteriosclerosis leads to myocardial infarction or cerebral infarction, it will also affect their lifespan, but there is no absolute conclusion.

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Can atherosclerosis be cured?

Atherosclerosis is a very common disease in daily life. Many patients, after being diagnosed with atherosclerosis, often ask doctors "Can my atherosclerosis be cured?" The answer is no, as atherosclerosis cannot be completely cured. However, this does not mean that atherosclerosis is untreatable. From a medical perspective, controlling blood pressure, lipids, and blood sugar, maintaining good lifestyle habits, exercising regularly, quitting smoking, controlling weight, and following a doctor's guidance to properly take medications can effectively control the onset and progression of atherosclerosis.

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What medicine is used for arteriosclerosis?

Commonly, some medications used to adjust blood lipids are employed, primarily to lower low-density lipoprotein levels, and some are also aimed at achieving target levels for triglycerides. These include statins, fibrates, and niacin. For some patients, antiplatelet drugs might be necessary to prevent acute thrombus formation and avoid acute incidents. For those who have already formed a thrombus, thrombolytic drugs can be used within a certain time window for some patients. For patients with coronary artery accumulation leading to angina, vasodilators, receptor blockers, or calcium channel antagonists might be used to alleviate the symptoms of angina.

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The difference between arteriosclerosis and atherosclerosis

Arteriosclerosis is the most common and most important among a group of vascular diseases called arteriosclerosis. The common characteristics of various arteriosclerosis include thickening, hardening, loss of elasticity, and narrowing of the arterial walls. The characteristic of arteriosclerosis is that the affected arterial lesions start from the intima, with various lesions coexisting, including local accumulation of papyraceous and complex carbohydrates, proliferation of fibrous tissue, and formation of plaques due to calcification, along with gradual degradation of the arterial media. Secondary lesions include intraplaque hemorrhage, plaque rupture, and local thrombosis formation. Modern cellular and molecular biology techniques show that arteriosclerotic lesions are characterized by migration of macrophages, proliferation of smooth muscle cells, and abundant formation of fibrous, collagen, elastic fibers, and proteoglycans as connective tissue matrices, as well as intra- and extracellular lipid accumulation. Because the lipid accumulation in the intima appears yellow and mushy, it is called arteriosclerosis. Although arteriosclerosis is only one type of arteriosclerosis, it is commonly referred to simply as arteriosclerosis due to its frequent occurrence and significant clinical relevance.