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Xie Zhi Hong

Cardiology

About me

The Secretary-General and Standing Committee Member of the Cardiovascular Disease Professional Committee of the Rehabilitation Medical Association in Ganzhou City, and a member of the Ganzhou City Working Committee of the Heart Emergency Branch of the China Medical Health International Exchange Promotion Association. He has chaired 4 provincial and municipal-level research projects, participated in more than 20 research projects, and published over 20 papers.

Proficient in diseases

Specializes in the diagnosis and treatment of coronary heart disease, hypertension, and structural heart disease. Research interests include hypertension, coronary heart disease, arrhythmia, and cardiac rehabilitation.

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Written by Xie Zhi Hong
Cardiology
51sec home-news-image

Can people with mitral valve regurgitation drink coffee?

The mitral valve refers to an important passageway between the atrium and the ventricle in the left heart system. This passageway is unidirectional. When the blood from the ventricle can flow back to the atrium through the mitral valve, it indicates mitral valve regurgitation. Mild to moderate mitral valve regurgitation often has no symptoms, and drinking coffee is permissible. However, patients with severe mitral valve regurgitation may experience severe palpitations, chest tightness, and shortness of breath. Drinking coffee can cause arrhythmias, palpitations, chest tightness, and increased heart workload; therefore, it is advised that such patients should not consume coffee.

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Written by Xie Zhi Hong
Cardiology
1min 56sec home-news-image

Pulmonary Arterial Hypertension Mild, Moderate, Severe Classification

Pulmonary artery pressure refers to the average pressure in the pulmonary artery when the body is at rest. Generally, the normal average pulmonary artery pressure is less than 25mmHg. Pulmonary hypertension is considered when the pulmonary artery pressure exceeds 25mmHg. The grading is generally divided into three levels based on the data: the first level is mild pulmonary hypertension with pulmonary artery pressure between 26-35mmHg; moderate with pressure between 36-45mmHg; and severe with pressure greater than 45mmHg. If classified by the functional impact of pulmonary hypertension, grade I pulmonary hypertension is characterized by patients who are not limited in daily activities, and do not experience symptoms such as shortness of breath, difficulty breathing, or chest tightness after physical activity. Grade II pulmonary hypertension patients are limited by mild activities, do not feel discomfort at rest, but experience symptoms like difficulty breathing, fatigue, chest tightness, and chest pain during everyday activities. Grade III pulmonary hypertension patients have significantly limited activity; they feel no discomfort at rest, but experience symptoms like difficulty breathing, fatigue, and chest pain after only mild activity, less than everyday activities. Grade IV pulmonary hypertension is severe, with patients experiencing symptoms such as difficulty breathing and feeling tightness in their chest even at rest without any activity.

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Written by Xie Zhi Hong
Cardiology
42sec home-news-image

Can rheumatic heart disease patients exercise?

Patients with rheumatic heart disease should go to the hospital for assessment, undergo cardiac ultrasound, and perform exercise tests. If the exercise test indicates cardiac function is below class II, or if the six-minute walk test reaches above class IV, exercise can be pursued. If patients have concerns, they can exercise under the guidance of a cardiac rehabilitation therapist at the hospital. For patients with cardiac function class III and above, exercise is not recommended. It is advised that they manage heart failure in the hospital or consider exercise only after surgery and when the condition is stable.

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Written by Xie Zhi Hong
Cardiology
40sec home-news-image

Why is there a murmur during diastole in mitral stenosis?

The mitral valve is a one-way piston valve between the left atrium and the left ventricle. Generally, once the ventricle completes a contraction and ejects blood, it immediately begins to relax, at which time the mitral valve opens to allow blood from the atrium into the left ventricle. When the mitral valve is severely narrowed, the blood from the atrium cannot pass through the mitral valve into the left ventricle in a timely manner. At this time, the blood flow speed at the mitral orifice increases, forming turbulence, which results in the appearance of diastolic murmurs.

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Written by Xie Zhi Hong
Cardiology
42sec home-news-image

Is rheumatic heart disease related to dampness?

Rheumatic heart disease often occurs in the southern regions where there is a higher level of humidity. Some patients develop rheumatic heart disease after suffering from rheumatic arthritis or other rheumatic conditions. In Western medicine, it is believed to be due to a series of immune system damages caused by infection with Group A streptococcus, leading to rheumatic inflammation of the heart or damage to the valve functions. This often presents as mitral stenosis, atrial fibrillation, or pericardial effusion. Therefore, according to traditional Chinese medicine theory, rheumatic heart disease is related to humidity.

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Written by Xie Zhi Hong
Cardiology
1min 12sec home-news-image

Is hyperthyroid heart disease dangerous?

Hyperthyroidism can repeatedly cause an increase in heart rate and metabolism, which is quite active, causing a long-term overload of heart function, leading to the formation of hyperthyroid heart disease, a chronic pathological change process. In the middle and early stages, it generally does not pose a life-threatening risk, only manifesting symptoms such as palpitations, tightness after activity, breathlessness, or chest tightness, which can improve with rest. However, if hyperthyroidism is not cured for a long time, or if hyperthyroid heart disease is not treated in time, it can lead to severe heart failure. If there is a severe decline in heart pump function, causing the ejection fraction to be below 30%, it can easily cause sudden pump failure or severe arrhythmias, such as ventricular tachycardia or fibrillation, posing a life-threatening risk to the patient.

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Written by Xie Zhi Hong
Cardiology
52sec home-news-image

Why does mitral valve insufficiency cause left ventricular hypertrophy?

The mitral valve is an important structure within the left ventricular system; it serves as the necessary channel for blood from the left atrium to enter the left ventricle. Normally, this channel allows for unidirectional flow. However, when mitral valve insufficiency occurs, blood flows back from the left ventricle to the left atrium. This leads to an increase in cardiac work, reduces the efficiency of the heart’s function, and causes hypertrophy due to overuse of the left ventricle. When the mitral valve closes to a certain extent, it can lead to an enlargement of the left ventricle. This is why mitral valve insufficiency can cause enlargement of the left ventricle, primarily because it increases ineffective work, leading to an increased cardiac load.

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Written by Xie Zhi Hong
Cardiology
1min 14sec home-news-image

The difference between sudden cardiac death and myocardial infarction.

Sudden cardiac death refers to the sudden death of a patient caused by various heart diseases. The most common causes include malignant arrhythmias such as ventricular fibrillation and ventricular tachycardia, severe bradycardia, significant valvular dysfunction, or the sudden occlusion of the coronary arteries leading to death. Myocardial infarction, on the other hand, refers to the sudden cessation of blood supply to the heart vessels, causing myocardial death and leading to arrhythmias or valvular dysfunction, which presents a range of symptoms in patients. Myocardial infarction can result in death in 30% of patients, but with medical intervention, the mortality rate can significantly decrease. Therefore, in cases of sudden cardiac death, the patients are already deceased. However, the mortality rate from myocardial infarction under natural circumstances is relatively high, reaching 30%. With current medical interventions, the rate of mortality due to sudden cardiac death can be reduced to less than 10%.

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Written by Xie Zhi Hong
Cardiology
1min 7sec home-news-image

How to deal with a cold and fever in rheumatic heart disease?

Patients with rheumatic heart disease who catch a cold and develop a fever may experience a worsening of their existing rheumatic heart disease, with more severe mitral stenosis or regurgitation, and there could also be recurrent attacks of existing heart failure. Therefore, if a person with rheumatic heart disease catches a cold, it is crucial to seek medical treatment early and go for a hospital examination. If it is a viral infection, antiviral treatment should be given, and for bacterial infections, medications to control and kill bacteria should be administered. Furthermore, if there are repeated fevers during the treatment process, physical methods to reduce fever can be used, such as applying a cold cloth or ice pack to the head when the temperature is below 38°C, along with medication therapy using ibuprofen. In short, it is essential to treat colds and fevers early in patients with rheumatic heart disease. (Medication should be used under the guidance of a physician.)

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Written by Xie Zhi Hong
Cardiology
1min 1sec home-news-image

The cause of thromboembolism in mitral stenosis

The mitral valve is an important part of the left heart system, located between the left ventricle and the left atrium. When the heart is in diastole, the mitral valve opens, allowing blood from the atrium to be transferred into the ventricle. However, when the mitral valve is narrowed, blood from the left atrium cannot flow into the left ventricle in a timely manner. This leads to an increased load on the left atrium. Over time, this can cause severe enlargement of the left atrium and lead to atrial fibrillation, a type of cardiac arrhythmia. Once atrial fibrillation occurs, it can cause thrombosis in the left atrium, particularly in the part called the atrial appendage. If a thrombus forms, it may dislodge during the heart's contractions, potentially causing systemic embolism with serious consequences, including paralysis or even sudden death.